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The CISD1 protein and CISD2 protein, which are present in mitochondria and the endoplasmic reticulum (ER), inhibit ferroptosis by reducing iron uptake from mitochondria and ROS production, respectively [57, 58]

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However, it is well known that mitochondrial and cytosolic GSH levels are decoupled during cell death and many other biological processes 40

18 It further bolsters bone health by working directly on the membranes of muscle cells to speed and enhance healing

A preliminary study unveils CISD2 as a ferroptosis-related therapeutic target for recurrent spontaneous abortion through immunological analysis and two-sample mendelian randomization