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Nakanishi M, Nomura J, Ji X, Tamada K, Arai T, Takahashi E, et al
doi: 10.1038/srep37744 (2016)
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The upsurge of very-low-density lipoprotein particles seems to be the core atherogenic dyslipidemia in T2DM, which is partially secondary to insulin resistance [26]
(21-23) Similarly, studies of humans to determine the risk of developing cirrhosis from alcohol have concluded that 50% of the phenotypic variation in terms of development of liver disease can be attributed to genetic factors
However, newer compounds are expanding the field by targeting multiple biological pathways simultaneously
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