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Erythrocyte adaptive metabolic reprogramming under physiological and pathological hypoxia

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Induces G2/M cell cycle arrest by inducing thiol oxidative stress resulting in -tubulin S-glutathionylation-mediated microtubule depolymerization

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Interestingly, the activated fibroblast phenotype, which is more proliferative and pro-inflammatory, is particularly pronounced in OA patients with obesity [88,89,90], even in non-load bearing joints i.e

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