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Thus, the transition from crystal deposition to acute inflammation can be conceptualized as a two-hit process: First hit: MSU crystal deposition on cartilage surfaces activates synovial fibroblasts to release CCL2/MCP-1, recruiting monocytes and establishing subclinical inflammation
Severyanova, L
Cardiovascular safety trial demonstrating that the DPP-4 inhibitor alogliptin does not increase MACE in patients with T2DM who had a recent acute coronary event
ACS Nano 17 , 1068910700 (2023)
It contains glycine, cysteine and glutamic acid
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