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In a homogeneous phase which is effective only for biocatalysis in which enzyme and substrate are soluble [70, 71]

Importantly, some identified genes fall outside established GLP-1 mechanisms, suggesting novel biology

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KAT8-mediated H4K16ac modification at these chemokine promoters, coupling with increased chromatin accessibility, facilitated the production and secretion of pro-inflammatory chemokines CXCL2 and CCL3 for neutrophil chemotaxis, neutrophil extracellular traps (NETs) formation and aggravated inflammatory damage in psoriasis