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It represents a fundamentally different absorption mechanism, one that bypasses the gastric destruction pathway entirely through phospholipid membrane fusion
Dieguez, C
We attempted to fully antagonize the extensive toxicity caused by NSAIDs (using diclofenac as a prototype)
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Developing therapeutic strategies to inhibit TIM-3 signaling could restore T cell activity and enhance immune efficacy against the inflammation and tissue damage characteristic of COPD (133)
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