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Rapid Communications in Mass Spectrometry, 2007
Fibroblasts have estrogen receptors on their surface and when being stimulated, they can increase collagen production [18]
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DHHC5 then catalyzes the palmitoylation of autophagy-related protein 9 A (ATG9A) at Cys155 and Cys156 [134], which drives the trafficking of ATG9A vesicles from the trans-Golgi network to the autophagosome nucleation site, providing the essential membrane source for the PI3P platform generated by the VPS34 complex [135]
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Pharmacologically targeting fatty acid synthase-mediated de novo lipogenesis alleviates osteolytic bone loss by directly inhibiting osteoclastogenesis through suppression of STAT3 palmitoylation and ROS signaling
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