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The primary actions of GLP-1 include stimulating insulin secretion from pancreatic beta cells in response to elevated blood glucose (importantly, this insulin release is glucose-dependent, reducing hypoglycaemia risk), suppressing glucagon release (which prevents the liver from producing excess glucose), and slowing gastric emptying
doi:10.1164/arrd.1983.127.4.504 Gay PC
This study noted that animals pretreated with CRE and CRE-AgNPs responded with better resumption in the activity of GST than irradiated animals not treated with the C
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Caloric restriction prevents age-associated accrual of oxidative damage to mouse skeletal muscle mitochondria
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