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A critic to the use of a murine model deficient for intestinal GCG products, would be that other gut hormones might compensate for the lack of a functional GCG gene in that tissue, hence explaining the normalized incretin effect
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GLP-1 therapies have emerged as an important innovation in addressing the global obesity challenge
[2] Oxidation-prone residues: peptides containing Cys/Met/Trp can be more susceptible to oxidation
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