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Programmed cell death in mature erythrocytes: a model for investigating death effector pathways operating in the absence of mitochondria
It is accepted that an excessive burden of ROS may lead to abnormal cell growth, corresponding changes in intracellular homeostasis and damage to important components of in the cell when the antioxidant defense and/or DNA repair mechanisms cannot balance the excess oxidants [29]
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This is generally mild and transient [13]
Bn cnh , ng qun kt hp ch n ung lnh mnh, ng gic v s dng kem chng nng hng ngy bo v da ton din
As natural vesicles secreted by autologous cells, Exo have low immune rejection and can transmit biological genetic information through blood-brain barrier [15]
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