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This can result in hypoglycemia or mitochondrial dysfunction
Interestingly, the thyroid profile went to normal when the glutathione drug administration was stopped
These include prostaglandins and leukotrienes that serve as signaling molecules during inflammation
Two possible roles have been suggested: biotransformation of electrophilic compounds, protecting the DNA content, where the GST catalyzes the conjugation of glutathione to the xenobiotic compound in a detoxification-related mechanism, and a possible role in modulating gene expression
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In addition, I-B-Kinase (IKK) complex is the core component of NF-B and is a target of ROS, affecting thus NF-B and S-glutathionylation and inhibited IKK activity (Reynaert et al
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