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Instead of single-gene resistance mechanisms, molecular investigations of persister cells indicate coordinated transcriptional and metabolic reprogramming (Lewis, 2012)

P1 was then gated on a plot of side-scatter width by side-scatter height around the clustered population (P2)

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Consistent with these facts, our assays demonstrate a more efficient inactivation of AX by DTT than with either GSH or N-acetyl-cysteine ( Table 2 )

Targeting NF-B: a promising molecular therapy in inflammatory arthritis