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TGF-1 also positively regulated the extracellular matrix (ECM) and led to significant up-regulation of the ECM marker proteins alpha smooth muscle actin (SMA) and zinc finger E-box-binding homeobox protein1(ZEB1) and down-regulation of E-cadherin, which in turn encouraged renal mesangial fibrosis and glomerulosclerosis[39]

The effect on appetite is partially mediated by the activation of neuropeptide Y (NPY) release in the hypothalamus