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Therapeutic strategies that target selective autophagy in the kidney will require carefully timed and precise cell-specific modulation, as well as biomarker-guided patient stratification, to improve efficacy and avoid adverse effects

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In hepatocytes, ATP7B plays a crucial role in facilitating the p62-mediated release of copper from intracellular stores into the cytoplasm, thereby enabling the excretion of surplus copper through its incorporation into bile (33)