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Tumors with HRD rely on alternative DNA repair mechanisms for survival, so agents that inhibit DNA single-strand break (SSB) repair or induce more SSBs trigger genomic instability and apoptosis 34
These pathways are posited to play a role in communicating stress signals and preparing the cell to counteract the incoming damage
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It delays AILI, and pathways associated with type I IFN production (Ifnb and Ifna4) are specifically upregulated in liver non-parenchymal cells [236]
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