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Dysregulation of these tight junctions contributes to a wide range of neurological disorders, including neurodegenerative diseases and neuroinflammation, thus underscoring their importance in CNS homeostasis
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The increase in energy expenditure could be explained by enhanced levels of PPAR-, PGC-1, and uncoupling protein-1 (UCP-1) in BAT as well as elevated levels of proopiomelanocortin in the hypothalamus
This more favorable hormonal milieu explains why GLP-1 weight loss tends to preserve more muscle mass, produce less hunger and psychological distress, and create a more sustainable long-term trajectory than equivalent weight loss from dieting alone
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Understanding your genetic predispositions in these pathways may help your provider tailor dosing, timing, or consider alternative GLP-1 agents
