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Drug Metab Pharmacokinetics 2014;29(5):367-72

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Chronic mitochondrial ROS production in HF further drives myocyte apoptosis via ROS-mediated activation of caspases and mPTP opening, fibrosis through ROS-activated TGF- signaling in fibroblasts, and electrical instability due to oxidative damage to ion channels, increasing arrhythmia risk

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Neurological Effects : AOD 9604 peptide shows minimal neurological side effects compared to compounds that cross the blood-brain barrier more readily