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J.CowanS

In this paper, we explore the potential role of certain amyloidogenic proteins, including amylin, amyloid beta, the prion protein, huntingtin, and -synuclein, in disease processes that result in the accumulation of deposits of protein fibrils, along with lipid membrane components of damaged mitochondria, which we argue may be a mechanism to sequester deuterium in order to reduce the deuterium burden in the tissues

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To further explore the relationship between ferroptosis and renal fibrosis in vitro, we detected fibrotic markers (fibronectin, -SMA and collagen-1) in TGF--induced HK-2 cells

Sensors 6, 756782

This syndrome is believed to be influenced by elevated levels of IL-6 and IL-10 [183]