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Suppression of Pdx-1 perturbs proinsulin processing, insulin secretion and GLP-1 signalling in INS-1 cells

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Various mechanisms may underlie the inhibitory effect of melatonin on tau hyperphosphorylation, as depicted in Fig

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cells play an active role in disease progression, as they express many T1D-associated genes, upregulate stress and inflammatory programs, and increase immune visibility under inflammatory or viral stress

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