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ME/CFS and Genetics: Chronic Fatigue Syndrome Genes, Immune Pathways, and Natural SolutionsGenetic variants in immune, mitochondrial, and inflammatory pathways (NK cells, NLRP3, interferon, complement, TRP channels) increase ME/CFS susceptibility
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In the mold phase, the TmGst1 showed the highest fold increase, being upregulated by almost threefold when compared to untreated samples (Fig
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