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rather, it reveals genetic predispositions in metabolic and peptide pathways
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Continue for the full 12-week timeline before concluding the serum isn't working
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(2013) recently demonstrated that DNA damage itself (i.e., produced by topoisomerase inhibitors) can promote HSV-1 replication in neuronal SH-SY5Y cells, suggesting again that the activation of DDR is required for a productive HSV-1 infection and it is abrogated during quiescence
Brain 2016
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