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Although redressing the BA composition has proved to be beneficial in mouse models, it was unlikely to translate to human pathophysiology, as changes in BA composition in mice hardly reflect similar changes in BA profiles in humans due to the differences in BA composition 41
Children and adolescents undergo significant metabolic and physiological changes due to growth and biological maturation that can increase ROS production, which may be accompanied by oxidative stress if levels of endogenous antioxidants are insufficient 14 , 23 , 33
, 30 , 42-59, doi: Lien, E
Specific knockdown of ERO1 can significantly alleviate ER oxidative stress caused by palmitic acid, reduce the accumulation of H 2 O 2 in the lumen and mitochondria, restore ER calcium homeostasis, and improve mitochondrial membrane potential and ATP levels, thereby effectively inhibiting palmitic acid-induced -cell death
