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Increased lactate concentrations are also intriguing in the context of one of the influential hypotheses on the provenience of ME/CFS symptoms, a state of chronic, low-level neuroinflammation [61]

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[9] Following oral administration, melatonin undergoes first-pass hepatic metabolism primarily through cytochrome P450 enzymes, particularly CYP1A2, resulting in the formation of 6-sulfatoxymelatonin, which is subsequently eliminated through renal excretion