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Discussion Our study establishes that non-gene-edited hCiPSC-NSCs, delivered via a dual-pathway strategy (intravenous systemic + intracerebroventricular boost), concurrently ameliorate ASD-like phenotypes in VPA-exposed rats by targeting the gut-brain axis

Ferroptosis: role of lipid peroxidation, iron and ferritinophagy

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Clinical, diagnostic, and therapeutic aspects of growth hormone deficiency during the transition period: review of the literature

To tackle this issue, we started by examining the physiology of BDNF-TrkB signalling in iSPNs, the most vulnerable neurons in HD