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In airway epithelial cells and alveolar macrophages exposed to CSE, PINK1-Parkin-mediated mitophagy enhances the clearance of damaged mitochondria, reduces mitochondrial reactive oxygen species (mtROS) and mtDNA leakage, thereby weakening the second signal of the NLRP3 inflammasome, inhibiting Caspase-1 activation and GSDMD-mediated pyroptosis (197, 198)

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