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There is growing evidence that disturbances of mitochondrial function, including increased oxidant production and altered mitochondria dependent signaling, contribute to the pathogenesis of vascular disease in atherosclerosis and other cardiovascular risk factors

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This disruption can result in cellular dysfunction, oncogenic transformation, senescence, aging, and programmed cell death, thereby hastening tissue degeneration and impacting overall health

One of them showed that a patient with level 1 anaphylaxis according to Brighton Criteria due to exenatide and lixisenatide was able to tolerate liraglutide without any problems [4, 7]