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Androgens stimulate periosteal bone formation in both men and male rodents [41, 42] and are known to have a direct effect on bone healing, as it induced callus formation in segmental bone defect mouse models [43], which are in line with our present results that male mice lacking AR (AR -/Y ;Prrx1::Cre) have decreased periosteal cell activity with low levels of collagens and integrins and testosterone can only promote bone fracture repair in AR flox/Y control mice, but not in AR -/Y ;Prrx1::Cre mice

This was seen in a roughly dose dependent manner but only led to 3 patients dropping out because of it