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Description

Unfortunately Phase III clinical trials of these drugs were prematurely terminated when some mild-to-moderate AD patients evidenced reduced cognitive functioning as compared with patients receiving placebo [51,52]

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Results Aminoglycosides and fluoroquinolones induce membrane damage and cytoplasmic condensation in E

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It may therefore interfere with methylation- dependent metabolism and indicate poor flow through the methionine cycle

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In contrast to the membrane disruption of red blood cells and Heinz bodies formation in favism, G6PD deficiency causing jaundice is perhaps attributed to the disruption of oxidant-antioxidant balance, impaired recycling of peroxiredoxin 2, thus affecting bilirubin clearance

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