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In 2009, the first unimolecular gut hormone receptor co-agonist, a glucagon/GLP-1 receptor co-agonist, was discovered and validated in preclinical models [76]
Those effects were partially independent of weight loss
The pivotal role of DPP-4 in endogenous GLP-1 metabolism was highlighted using the isolated perfused porcine small intestine, which revealed that over half of newly released GLP-1 was degraded even before it left the splanchnic bed
While not the main focus of the stack, these heart benefits add research value for studies of systemic health
The mechanism typically involves IgE-mediated hypersensitivity, particularly with exenatide-based medications that have a non-human peptide origin
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