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Figure EV2 Ventricular weight and echocardiography in vehicle- and -MSH-treated mice after 8 weeks of sham operation

Moreover, increased levels of LCACs may also induce endoplasmic reticulum (ER) stress and accelerate the progression of cell damage [30]

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On the basis of these observations, we hypothesize that excessive H 2 O 2 , if unresolved in time, leads to irreversible severe astrogliosis and chronic oxidative stress that further depletes astrocytic Hb, perpetuating a vicious cycle to accelerate AD progression (Supplementary Fig