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In an in vitro/ex vivo study, treatment of ApoE / mice with liraglutide for 4 weeks dose-dependently inhibited angiotensin II-induced VSMC proliferation by activating AMPK signaling and inducing cell cycle arrest, thus delaying the progression of atherosclerosis, independently of its glucose-lowering effect [52]

Exposure to environmental toxins or heavy metals

Translationally, these data nominate formate as a tractable SCFA-class metabolite that does not primarily act via FFAR2/3 or HDAC but instead fuels nucleotide biosynthesis in T cells [52]

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