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doi: 10.3389/fneur.2020.578003 87 Blank-SteinNMassE
The primary mechanisms we see cited in the literature include: VEGF upregulation: BPC-157 increases vascular endothelial growth factor, which drives new capillary formation in injured tissue
Furthermore, -MSH analogs like Melanotan 1 appear to downregulate the mRNA expressions of liver transforming growth factor 1 (TGF-1), collagen 1, and various cell adhesion molecules, which are posited to be key players in the progression of fibrosis
When Treatment Plans Change Your lab results help your doctor make decisions about your care
D., Wadden
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