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CD36 is considered a biomarker for atherosclerotic lesion progression since it stimulates inflammatory response and foam cell formation
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(B) Western blot of endogenous A39 and ectopically expressed FLAG-tagged A39 WT , substrate binding pocket mutant A39 C334S , and matrix loop 1 four cysteine mutant A39 4CA in the K562 cells upon GSH depletion by BSO treatment (1mM, 2d)
Importantly, the mere presence of PanIN lesions is not necessarily problematic, as low-grade PanINs are present in 50-80% of adults and do not universally transform into malignancy 21
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