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Glutamatergic theories of schizophrenia are based on the ability of N-methyl-D-aspartate receptor (NMDAR) antagonists to induce schizophrenia-like symptoms, as well as emergent literature documenting disturbances of NMDAR-related gene expression and metabolic pathways in schizophrenia

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Under physiological conditions, p53 acts to sustain the cardiac architecture and adjust the levels of transcripts correlated with metabolism, the cardiac architecture, and excitationcontraction coupling

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