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The impaired GLP-1 response observed in obesity and type 2 diabetes has, therefore, been implicated in the pathogenesis of hyperglucagonaemia [63]
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Specifically, it acts as a single peptide that simultaneously activates three receptors involved in metabolic regulation: the glucose-dependent insulinotropic polypeptide receptor (GIPR), the glucagon-like peptide-1 receptor (GLP-1R), and the glucagon receptor (GCGR)
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G., Brouwers, O
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