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In conclusion, these findings demonstrate that a clinical intervention for 3-months with oral L-GSH in individuals with T2DM can decrease levels oxidized GSH, while maintaining levels of reduced GSH and overall reducing the extent of oxidative stress

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A threshold that doesn't trigger clinical deficiency symptoms but correlates with persistent fatigue, brain fog, and impaired fat metabolism

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The cumulative urinary excretion of the parent drug amounted to only 3.01%

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