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The impact of the immune-inflammatory response during acute COVID-19 on the neuropsychiatric symptoms of Long COVID is partially mediated by OSTOX/ANTIOX
Small molecules that substitute for peptides at receptor targets are emerging in pharmaceutical pipelines, assisted in design by advanced imaging technologies
p =0.020) but not AA genotype (OR 1.23 [95% CI 0.26, 5.94]
In Covid-19, ED (a hallmark of the disease) and activation of adhesion molecules trigger the progression of immunothrombosis and the development of critical complications (Bonaventura et al
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