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Activation of human microglia by fibrillar prion protein-related peptides is enhanced by amyloid-associated factors SAP and C1q
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The resulting, locally elevated concentration of pro-inflammatory molecules directly causes hepatocyte injury and apoptosis, for example via TNF-mediated death receptor activation (169), initiating and perpetuating a vicious cycle of necroinflammation leading to loss of hepatocytes and of the livers functional capacity (170)
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