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[4] The scientists note that over the course of the study, VIP would emerge as an endogenous modulator of pulmonary vascular remodeling and inflammation, through its suppression of NFAT activation. In particular, NFAT suppression may play an important role in preventing pulmonary fibrosis, the end stage of diverse inflammatory conditions such as COPD, sarcoidosis, etc
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Both verified at 1%
10.1073/pnas.0407162101 50 EpelE
Cells 11(16):2617 Laval L, Martin R, Natividad J, Chain F, Miquel S, de Maredsous CD et al (2015) Lactobacillus rhamnosus CNCM I-3690 and the commensal bacterium Faecalibacterium prausnitzii A2165 exhibit similar protective effects to induced barrier hyper-permeability in mice
The samples were sent to Wuhan Servicebio Biotechnology Co
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