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What makes it special is that it is designed to disrupt the interaction between FOXO4 and the tumor-suppressor protein p53 within senescent cells, thereby triggering programmed cell death (apoptosis) of those cells

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The chemistry of reactive oxygen Species (ROS) revisited: outlining their role in biological macromolecules (DNA, lipids and proteins) and induced pathologies

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The mechanistic case is coherent and supported by what we know about each modality separately

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