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Indeed, genetic deficiency of the translocase markedly reduced the mitochondrial acetylcarnitine-dependent nuclear histone acetylation, indicating the significance of the carnitine-dependent mitochondrial acetyl group contribution to histone acetylation
Co-immunoprecipitation mass spectrometry and CRISPR knockout (KO) in mammalian cells identified mitochondrial m-AAA protease AFG3L2 as responsible for degrading SLC25A39 through the matrix loop 1
Andersen, E.S
It's an essential component in energy generation, as it carries long-chain fatty acids into the mitochondria (your cell's energy center) where the fats are oxidized (i.e
However, the mechanisms driving this increased efficacy remained unclear
Available from: Biaggioni I (2024): Adrenoceptor Agonists & Sympathomimetic Drugs (Chapter 9)
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