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The question of how in the presence of adequate PUFA precursors the constitutive and inducible forms of prostaglandin H synthetase can be induced to generate the appropriate forms of prostaglandin required to maintain tissue homeostasis will rely on further understanding of the co-factors and feedback mechanisms involved

Clinical monitoring should include: Periodic assessment of liver enzymes (ALT, AST) to detect transient increases

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This can be understood as a self-perpetuating triad: Immune Dysregulation: The immune system mistakenly targets and attacks thyroid tissue