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This is the mechanistic basis for the higher GI discontinuation rate seen with CagriSema at maximum doses
This allows the nutrient to concentrate in tissues and then go where its needed the most
Please do not just go to a semaglutide or GLP-1 "party!" What exactly is semaglutide and how is it used
Activation of the GLP-1 receptor leads to inhibition of gastric emptying and small bowel motility leading to a slower absorption of nutrients
Where they differ is in which receptors they target most strongly and that determines their primary effects
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