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Serving Size 2 Capsules Servings Per Container 30 Vitamin D3 (as cholecalciferol) 50 mcg (250% DV) Vitamin B6 (as pyridoxal 5'-phosphate) 26 mg (1,529% DV) Folate (from L-5-MTHF-Ca) 400 mcg DFE (100% DV) Vitamin B12 (as methylcobalamin) 525 mcg (21,875% DV) Biotin 5 mcg (17% DV) Iron (as ferrous bisglycinate chelate) 18 mg (100% DV) Magnesium (as magnesium oxide & citrate) 200 mg (48% DV) Zinc (as zinc bisglycinate chelate) 20 mg (182% DV) Digestive Comfort & Soothing Blend 325 mg ** Gingerols Ginger Root Extract (5% gingerols) Peppermint Leaf Extract Bromelain Powder (2,400 GDU/g) DigeZyme Multi-Enzyme Complex LactoSpore (Bacillus coagulans MTCC 5856) 166 mg (6B CFU) ** Energy & Vitality Complex 126 mg ** Ashwagandha Extract (root, 5% withanolides) Nutrient Replenishment Matrix 22 mg ** BioPerine Black Pepper Extract (fruit) 5 mg **

To view a copy of this licence, visit About this article Cite this article Snchez-Valle, J., Flores-Rodero, M., Costa, F.X

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The molecular basis of lutealization and progesterone synthesis is the presence of core steroidogenic genes (STAR, CYP11A1, LHCGR, and PGR) and transcriptional regulators (SF-1, FOXO1 and LRH-1), whereas endocrine signaling (MAPK/ERK, PI3K/AKT, and WNT/-catenin) fine-tune luteal cell survival, angiogenesis, In addition to classical hormonal regulation, new evidence also indicates that metabolic-epigenetic integration is central in the process of defining luteal fate, where AMPK can serve as an important sensor of energy, PPAR can be a context-specific transcriptional regulator of lipid metabolism and cell fate choices, and H3K27me3, which is mediated by EZH2, is a stable manner in which steroidogenic genes are repressed in luteal regression

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2e, g) in males or females