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GLP-1R triggers various intracellular signaling pathways, such as the cAMP/PKA and PI3K/Akt pathways, by binding to endogenous GLP-1 or exogenous agonists, resulting in the enhancement of insulin secretion, inhibition of glucagon secretion, as well as anti-inflammatory and antioxidant stress response
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For long term treatment it is wise not to exceed the dose beyond 2.6 gm/day
This, in turn, has a negative effect on wound healing
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That said, monitoring kidney function more frequently during tirzepatide initiation is prudent
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