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We observed that AOAA treatment decreases cysteine and GSH levels while increases ROS and ferroptosis stresses in SCD erythroblasts (Supplementary Fig
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10.1111/j.1365-2982.2010.01487.x 171
Indeed, glutathione deficiency is central to alcoholic liver disease (ALD), with a reduction in glutathione one sign of chronic alcohol consumption
all such literature is pre-clinical
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