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Fluoxetine ameliorates dysbiosis in a depression model induced by chronic unpredicted mild stress in mice (in Eng)
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Because it modulates a methylation and NAD+ pathway, interactions with other metabolically active compounds cannot be ruled out and have not been characterised in humans
The peptide simply does not interact with the receptors and pathways that stimulate IGF-1 production
Do not neglect these fundamentals while focusing on the peptide
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