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Thats because the tripeptide plays an important role in Phase II detoxification processes in the liver
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It describes a very large category of molecules
The impairment of astrocytic autophagy plasticity due to the loss of LC3B and SQSTM1 function accelerates A aggregation and increases GFAP-positive astrocytes in AD mice, thereby amplifying neuronal damage and memory deficits 13 (Fig
However, N-terminal modifications, such as N-acetylation, D-norleucine substitution, or replacement with -aminobutyric acid (GABA), dramatically elongated the peptides half-lives
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