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As GEP44 acts directly on muscle tissue, while its effects on islets require concomitant Y1-R antagonism, activation of Y1-R expressed in muscle tissue may ultimately prove to be a critical factor in reducing serum glucose levels, alongside any potential effects on insulin secretion and glucagon release

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This regulatory distinction is important because supplements are not subject to the same rigorous testing, quality control, and safety monitoring as prescription pharmaceuticals

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