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Different mechanisms are pro-posed to explain curcumin's effect on glucose homeostasis, which are mainly based on the activation of the gamma receptor, which is activated by the peroxisome proliferator (PPAR-y)

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The extension is not a rationally designed pharmacophore, it is a residual fragment of the methionyl porcine growth hormone fusion partner used to express the protein in Escherichia coli, characterised in a companion paper in the same journal and year